Ipsilateral facial weakness

Central facial palsy colloquially referred to as central seven is a symptom or finding characterized by paralysis or paresis of the lower half of one side of the face. It usually results from damage to upper motor neurons of the facial nerve. The facial motor nucleus has dorsal and ventral divisions that contain lower motor neurons supplying the muscles of the upper and lower face, respectively. The dorsal division receives bilateral upper motor neuron input i. Thus, lesions of the corticobulbar tract between the cerebral cortex and pons and the facial motor nucleus destroy or reduce input to the ventral division, but ipsilateral input i.
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Central facial palsy

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Differentiating Facial Weakness Caused by Bell's Palsy vs. Acute Stroke | JEMS

She complains of no pain or numbness. You perform a neurologic exam; strength and sensation are normal throughout, with no weakness in the arms or legs and no other neurologic findings. Is this a stroke? Anatomy of Facial Muscle Control Two facial nerves, the right and the left, control all of the muscles in the face. The right facial nerve controls all of the muscles on the right side and the left facial nerve controls all of the muscles on the left side of the face. The facial nerves emerge from the middle of the brainstem the pons and carry motor fibers to the muscles of facial expression. These fibers come from the motor cortex of both cerebral hemispheres.
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Patterns of pontine strokes mimicking Bell’s palsy

Facial nerve palsy is a neurological condition in which function of the facial nerve cranial nerve VII is partially or completely lost. It is often idiopathic but in some cases, specific causes such as trauma, infections, or metabolic disorders can be identified. Two major types are distinguished: central facial palsy lesion occurs between cortex and nuclei in the brainstem and peripheral facial palsy lesion occurs between nuclei in the brainstem and peripheral organs.
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Metrics details. Peripheral-type facial palsy very rarely arises from pontine stroke. We attempted to identify unique clinico-radiologic patterns associated with this condition. Patients with pontine tegmentum stroke and acute onset of peripheral-type facial weakness were reviewed from the acute stroke registry of a tertiary hospital. The clinico-radiologic patterns of 10 patients were classified into one of three types based on the respective stroke mechanism.
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